Protective effect of carnosic acid on pulmonary fibrosis: Antioxidant and antifibrosis effects of carnosic acid on pulmonary fibrosis
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The first in vitro part showed that CA induces apoptosis in HLF in a dose-dependent manner, accompanied by cell cycle arrest in the G0/G1 phase and activation of the expression of the signaling proteins Akt, p38-MAPK and p21. The second part confirmed the protective effect of this biomolecule in vivo on the fibrosis process and oxidative stress induced by BLM.
In the light of these results, it would be interesting to test the efficacy of CA on certain diseases whose physiopathology is dominated by inflammation, oxidative stress and cell proliferation. .
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