Mitocondriopatia- Arqueal/Mitocondrial Digoxina e Na-K ATPase: Síndrome de inibição da Na-K ATPase e disfunção mitocondrial
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A diminuição da disponibilidade de magnésio em consequência do aumento do cálcio provoca uma produção defeituosa de ATP mitocondrial que afecta a ATP sintase. A disfunção mitocondrial resulta na redução incompleta de O2 e no aumento da produção do radical livre, o ião superóxido.
O ião superóxido pode abrir o poro PT, completando o ciclo de destruição mitocondrial. A disfunção mitocondrial tem sido implicada na patogénese da degeneração neuronal, como a doença de Parkinson e o envelhecimento neuronal.
Os danos causados pelos radicais livres têm sido implicados na oncogénese, na degeneração neuronal e no envelhecimento. .
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