Aceruloplasminemia Clinical signs and pathophysiological mechanisms
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Patients most often show initial systemic signs, and then develop neuropsychiatric symptoms and cerebral iron overload. On the other hand, rare cases develop neuropsychiatric symptoms without subsequent systemic signs or cerebral iron overload.
These differences are explained by genetic characteristics, ceruleoplasmin levels and metabolic disorders. Clinicians should be alert to this disease in diabetic men and women with anemia, in the presence of neurological symptoms such as abnormal movements, epilepsy and ataxia, in order to propose appropriate management.
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£27.18
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